Søgning på udtrykket 'cvid' giver 9 resultater
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Denne vejledning er tiltænkt behandling af CVID hos voksne patienter. Der er ikke foretaget en gradering af evidensniveau, da der ikke foreligger solid evidens for flere dele af feltet.
Denne vejledning er tiltænkt diagnostik og kontrol af CVID hos voksne patienter. Der er ikke foretaget en gradering af evidensniveau, da der ikke foreligger solid evidens for flere dele af feltet.
Udarbejdet af Hanne V. Marquart, Carsten Heilmann, Terese Katzenstein, Carsten Schade Larsen, Klaus Müller, Thomas Hoffman, Helene Ingels, Lars P. Ryder, Thyge Lynghøj Nielsen, Niels Fisker, Jens Erik Veirum
19. maj, Online og Aarhus Universitetshospital
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Clinical & Experimental Immunology
Clinical & Experimental Immunology, EarlyView.
Current Opinion in Infectious Diseases
munodeficiency (CVID). High rates of CMV end-organ disease, mortality, development of CMV resistance and prolonged antiviral use have been observed in individuals with CVID. Summary We recommend that clinicians tailor their approach to the individual based on their underlying immune deficit and maintain a high index of suspicion and low threshold for treatment. More research is required to improve stratification of CMV risk in PID, develop new diagnostic tools and manage end-organ disease in this cohort.
Clinical Microbiology and Infection
A 22-year-old man with common variable immunodeficiency (CVID) complicated with granulomatous–lymphocytic interstitial lung disease (GLILD) previously treated with azathioprine and a 4-weekly course of rituximab 3 years before who was receiving subcutaneous (SC) immunoglobulin replacement therapy (IRT) was diagnosed of COVID-19 by SARS-CoV-2 reverse-transcriptase-polymerase-chain-reaction (RT-PCR) of a nasopharyngeal swab specimen after a 4-day history of fever. He quarantined at home but was later admit
BMC Infectious Diseases
viral mutations conferring antiviral drug resistance. Case presentation Here we reported the case of an HCMV seronegative patient with common variable immunodeficiency (CVID), multiple hepatic adenomatosis, hepatopulmonary syndrome and portal hypertension who received a liver transplant from an HCMV seropositive donor. The patient was treated with Valganciclovir (vGCV) and then IV Ganciclovir (GCV) at 5 week post-transpl
Clinical Infectious Diseases
mplification. The AQSA resistance phenotype (≥45% parasite survival) was expressed in 36.5% (23/63) of isolates and was significantly associated with treatment failure (P = .0020). Pfmdr1 mutant haplotypes were N86/184F/D1246, and Pfcrt was CVIET or CVIDT at positions 72–76. Additional Pfcrt mutations were not associated with amodiaquine resistance, but the G353V mutant allele was associated with ACPR compared to Pfmdr1 haplotypes harboring F1068L or S784L/R945P mutations (P = .030 and P = .